Pancreatic β Cell Dedifferentiation as a Mechanism of Diabetic β
Frontiers The role of GABA in islet function
SWELL1 depletion reduces swell-and glucose-stimulated β-cell
Contribution of Mitochondria to Insulin Secretion by Various
LRRC8/VRAC anion channels enhance β-cell glucose sensing and
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Depolarization of the pancreatic β-cell by glucose: the VRAC
Depolarization of the pancreatic β-cell by glucose: the VRAC
volume regulated anion channel
SWELL1 is a glucose sensor required for β-cell excitability and
CFTR and Anoctamin 1 (ANO1) contribute to cAMP amplified
Biomolecules, Free Full-Text